This site is intended for healthcare professionals

Go to /sign-in page

You can view 5 more pages before signing in

Functional hyposplenia

Last reviewed dd mmm yyyy. Last edited dd mmm yyyy

Authoring team

  • splenic hypoplasia is a poorly defined and infrequently recognized condition that is usually not associated with other anomalies and may be familial
  • functional asplenia is associated with conditions such as homozygous sickle cell disease, haemoglobin sickle cell disease, and sickle cell haemoglobin (HbS) beta thalassemia
    • most children with these hemoglobinopathies are functionally hyposplenic starting in the first year of life and become anatomically asplenic (due to splenic infarction and splenic atrophy) by the second decade of life
    • infection risks in these individuals parallel those of patients with asplenia
    • patients who undergo splenectomy because of thalassemia or Hodgkin disease have a higher risk of overwhelming infection than those patients with functional hyposplenia secondary to sickle cell disease
    • additional conditions associated with splenic hypofunction include neonatal age, rheumatologic diseases (systemic lupus erythematous (SLE), rheumatoid arthritis), inflammatory bowel disease, graft versus host disease, coeliac disease and nephrotic syndrome

Reference

  1. Kirkineska L et al. Functional hyposplenism. Hippokratia v.18(1); Jan-Mar 2014.

Create an account to add page annotations

Add information to this page that would be handy to have on hand during a consultation, such as a web address or phone number. This information will always be displayed when you visit this page

The content herein is provided for informational purposes and does not replace the need to apply professional clinical judgement when diagnosing or treating any medical condition. A licensed medical practitioner should be consulted for diagnosis and treatment of any and all medical conditions.

Connect

Copyright 2024 Oxbridge Solutions Limited, a subsidiary of OmniaMed Communications Limited. All rights reserved. Any distribution or duplication of the information contained herein is strictly prohibited. Oxbridge Solutions receives funding from advertising but maintains editorial independence.