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Aetiology

Authoring team

  • increased intake:
    • excessive vitamin D
    • infants on excessive amounts of high phosphate feeds
    • TPN with excessive phosphate concentration
  • decreased excretion:
    • renal failure, with or without secondary hyperparathyroidism
    • hypoparathyroidism
  • redistribution:
    • therapeutic destruction of cells in malignancy
    • reduced celluar uptake in insulin deficiency
  • acromegaly - hyperphosphataemia and hypercalcaemia may occur
  • acidosis - this causes a rise in intracellular phosphate

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